Agbl3, though specific aliases are not provided in the given references, likely has important functions within the body. While its exact function, associated pathways, and overall biological importance aren't detailed in the references, genetic models can be valuable for further study.

In one study, a family presented a small duplication in cis affecting AGBL3 along with CALD1. Four other patients carried larger deletions encompassing AGBL3 among other genes. All patients had neurodevelopmental delay, mild to moderate intellectual disability, dysmorphic features, and a behavioral phenotype of aggressiveness and disinhibition. This indicates AGBL3's potential role in neurodevelopment and behavior [1].

In another study, a SLC6A3-AGBL3 fusion event was detected in a meningioma tumor sample. Fusion genes in meningiomas are more likely in tumors with greater genomic instability or in patients with a history of cranial irradiation, suggesting AGBL3 might be involved in genomic stability-related processes in meningiomas [2].

In conclusion, AGBL3 seems to be associated with neurodevelopmental and behavioral aspects as well as potentially playing a role in genomic stability in meningiomas. These associations from the limited data available highlight the importance of further in-vivo studies, like gene knockout (KO) or conditional knockout (CKO) mouse models, to precisely define AGBL3's essential biological functions and its implications in related diseases.